Claim · model-written summary
Ethylene enhanced nitrate reductase activity.
- Taxon
- Vicia faba L
Vicia fabat0 - Treatment
- ethylenee0
- Gene
- nitrate reductasee0
- Outcome
- nitrate reductase activitye0
- Direction
- enhancee0
Not reportedMaterialCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“ethylene was shown to enhance nitric oxide levels and, corresponding, nitrate reductase activity”
t0“Vicia faba L”
Title
[NO may function in the downstream of Ca2+ in ethylene induced stomatal closure in Vicia faba L].
Guo Hua Liu; Jing Liu; Li Xia Hou; et al. PubMed 2009
Other claims from this paper (9)
- Intervention effect Ethephon caused time- and dose-dependent stomatal closure under light.
- Intervention effect The NO scavenger cPTIO reduced ethylene-induced stomatal closure.
- Intervention effect The nitrate reductase inhibitor NaN3 reduced ethylene-induced stomatal closure.
- Intervention effect Extracellular Ca2+ chelation with EGTA reduced ethylene-induced stomatal closure.
- Intervention effect Ethylene enhanced nitric oxide levels.
- Intervention effect Nitrate reductase inhibition diminished ethylene-induced NO production in guard cells and leaves.
- Intervention effect Compromising calcium reduced ethylene-induced NO levels.
- Intervention effect Compromising calcium reduced nitrate reductase activity.
- Mechanism Nitrate reductase-derived NO may act downstream of calcium in ethylene-induced stomatal movement.