Claim · model-written summary
Mutations in the miR167 target sites of ARF6 or ARF8 caused ectopic expression in ovule and anther domains where miR167 is normally present.
- Taxon
- Arabidopsis
Arabidopsise1 - Organ / stage
- domains of both ovules and antherse4
- Treatment
- Mutations in the miR167 target sites of ARF6 or ARF8e4
- Gene
- ARF6 or ARF8e4
- Outcome
- ectopic expressione4
- Direction
- increaseinferrede4
Not reportedMaterialCompoundInteractorInteractor roleSecond interactorSecond interactor roleComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Verbatim
e1“Two Arabidopsis Auxin Response Factors, ARF6 and ARF8, regulate gynoecium and stamen development in immature flowers”
e4“Mutations in the miR167 target sites of ARF6 or ARF8 caused ectopic expression of these genes in domains of both ovules and anthers where miR167 was normally present”
Title
Miin-Feng Wu; Qing Tian; Jason W. Reed Development 2006. doi:10.1242/dev.02602
Other claims from this paper (7)
- Association Wild-type pollen grew poorly in arf6 arf8 gynoecia.
- Association Poor pollen growth correlated with ARF6 and ARF8 expression in the style and transmitting tract.
- Intervention effect Overexpressing miR167 mimicked the arf6 arf8 phenotypes.
- Intervention effect The miR167 target-site mutations arrested ovule integument growth.
- Intervention effect The miR167 target-site mutations made anthers grow abnormally.
- Intervention effect The miR167 target-site mutations made anthers fail to release pollen.
- Mechanism miR167 is essential for correct patterning of gene expression and for fertility of ovules and anthers.