Claim · model-written summary
Hydrogen peroxide accumulation promoted NO production through nitrate reductase activation.
- Taxon
- Rice
Oryza sativat0 - Gene
- nitrate reductasee0
- Compound
- hydrogen peroxide (H(2)O(2))e1
- Outcome
- NO productione0
- Direction
- promotede0
Not reportedMaterialOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleTreatmentComparatorSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
e0“which consequently promoted NO production via the activation of nitrate reductase”
e1“increase of hydrogen peroxide (H(2)O(2)) in the leaves”
t0“Rice”
Title
Aihong Lin; Yiqin Wang; Jiuyou Tang; et al. PLANT PHYSIOLOGY 2011. doi:10.1104/pp.111.184531
Other claims from this paper (9)
- Observation An NO accumulation mutant called noe1 was identified in rice.
- Observation NOE1 encodes the rice catalase OsCATC.
- Observation The noe1 mutant increased hydrogen peroxide in leaves.
- Intervention effect Removing excess NO reduced leaf cell death in noe1 plants.
- Intervention effect Overexpressing GSNOR1 lowered SNO levels and alleviated noe1 leaf cell death.
- Observation S-nitrosylated proteins were identified in wild-type plants.
- Observation S-nitrosylated proteins were identified in noe1 plants.
- Observation Glyceraldehyde 3-phosphate dehydrogenase and thioredoxin were NO targets unique to noe1 plants.
- Mechanism The authors infer that NO and SNOs mediate hydrogen peroxide induced leaf cell death.