Paper · 2001 · Plant and Cell Physiology

Effect of Methyl Jasmonate on Harpin-Induced Hypersensitive Cell Death, Generation of Hydrogen Peroxide and Expression of PAL mRNA in Tobacco Suspension Cultured BY-2 Cells

Salamah Andi; Fumiko Taguchi; Kazuhiro Toyoda; et al.

3
claims
4
verbatim quotes
57
citations
3
research areas
✓
Open access

Abstract

Methyl jasmonate inhibited the harpin-induced defense responses such as cell death, H2O2 generation and gene expression encoding phenylalanine ammonia-lyase in tobacco suspension cultured BY-2 cells. These results suggest that MeJA may act as an endogenous suppressor for plant defense response including hypersensitive reaction.

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The text the model read, with the source record’s inline markup (italics, sub- and superscripts) rendered and its character entities decoded; quote offsets refer to the raw text.

Claims 3

Intervention effectobservedoriginal resultnegativemeasured

Claim · model-written summary

Methyl jasmonate inhibited harpin-induced cell death and hydrogen peroxide generation in tobacco BY-2 cells.

Material
BY-2e0
Treatment
Methyl jasmonatee0
Outcome
cell death, H2O2 generatione0
Direction
inhibitede0
Qualifier
harpin-inducede0

Not reportedComparator

Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Nicotiana tabacum
Nicotiana tabacum · TaxonJom / Joachim Müllerchen · CC BY 2.5 · Wikimedia Commons ↗

Verbatim

e0

“Methyl jasmonate inhibited the harpin-induced defense responses such as cell death, H2O2 generation and gene expression encoding phenylalanine ammonia-lyase in tobacco suspension cultured BY-2 cells.”

✓ located verbatim in the source · abstract, characters 0–199 · show in text ↑
Intervention effectobservedoriginal resultnegativemeasured

Claim · model-written summary

Methyl jasmonate inhibited harpin-induced phenylalanine ammonia-lyase gene expression in tobacco BY-2 cells.

Material
BY-2e0
Treatment
Methyl jasmonatee0
Gene
phenylalanine ammonia-lyasee0
Outcome
gene expression encoding phenylalanine ammonia-lyasee0
Direction
inhibitede0
Qualifier
harpin-inducede0

Not reportedComparator

Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Nicotiana tabacum
Nicotiana tabacum · TaxonJom / Joachim Müllerchen · CC BY 2.5 · Wikimedia Commons ↗

Verbatim

e0

“Methyl jasmonate inhibited the harpin-induced defense responses such as cell death, H2O2 generation and gene expression encoding phenylalanine ammonia-lyase in tobacco suspension cultured BY-2 cells.”

✓ located verbatim in the source · abstract, characters 0–199 · show in text ↑
Mechanismhypothesisoriginal resultnegativemeasured

Claim · model-written summary

MeJA may act as an endogenous suppressor of plant defenses including hypersensitive reaction.

Compound
MeJAe0
Outcome
plant defense response including hypersensitive reactione0
Qualifier
may act as an endogenous suppressore0

Organisms pictured · Representative pictures of the matched species, not figures from the paper.

Nicotiana tabacum
Nicotiana tabacum · TaxonJom / Joachim Müllerchen · CC BY 2.5 · Wikimedia Commons ↗

Verbatim

e0

“These results suggest that MeJA may act as an endogenous suppressor for plant defense response including hypersensitive reaction.”

✓ located verbatim in the source · abstract, characters 200–329 · show in text ↑
e1

“in tobacco suspension cultured BY-2 cells”

✓ located verbatim in the source · abstract, characters 157–198 · show in text ↑

(-)-methyl jasmonate · physiology: oxidative · Nicotiana tabacum (tobacco)Positive and negative results

4 papers: 3 positive, 1 negative or no effect

3 in the opposite direction

  1. positive
  2. positive
  3. positive
See all 4 papers →

(-)-methyl jasmonate · molecular: expression · Nicotiana tabacum (tobacco)Positive and negative results

13 papers: 6 positive, 7 negative or no effect

6 papers in the same direction as this paper · 6 in the opposite direction

  1. positive
  2. positive
  3. positive

9 papers more in this group

See all 13 papers →

Claims are grouped by organism, treatment and what was measured or targeted. A paper counts as positive when any of its claims in the group is positive.