Claim · model-written summary
Pretreatment with 1-methylcyclopropene abolished the monomeric G-protein response to ethylene.
- Taxon
- Pea
Lathyrus oleraceust0 - Treatment
- 1-methylcyclopropenee0
- Outcome
- response to ethylenee0
- Direction
- abolishese0
- Comparator
- ethylenee0
Not reportedMaterialGeneCompoundOrgan / stageInteractorInteractor roleSecond interactorSecond interactor roleSettingValueQualifier
Organisms pictured · Representative pictures of the matched species, not figures from the paper.
Verbatim
t0“Pea”
e0“Pretreatment with 1-methylcyclopropene abolishes the response to ethylene overall.”
Title
Igor E. Moshkov; Galina V. Novikova; Luis A.J. Mur; et al. PLANT PHYSIOLOGY 2003. doi:10.1104/pp.102.015057
Other claims from this paper (6)
- Intervention effect Ethylene caused rapid bimodal up-regulation, transient unimodal activation or transient down-regulation of monomeric G-proteins in pea epicotyls.
- Observation Immunoprecipitation suggested some affected monomeric G-proteins belong to the Rab class.
- Intervention effect Ethylene rapidly up-regulated protein kinase activity in pea epicotyls in a bimodal pattern.
- Intervention effect 1-methylcyclopropene inhibited the ethylene effect on pea protein kinase activity.
- Observation Immunoprecipitation indicated the responsive kinases are in the MAP kinase ERK1 group.
- Mechanism The authors propose a separate and antagonistic ethylene transduction chain.